MORE....Polio Information & Polio Vaccine Dangers
Stephan Cooter, Ph.D (taken from Beating Chronic Illness): Beriberi or Aseptic Spinal Meningitis? Getting shots is only one player in producing what I believe are the true causes behind Chronic illness. There are many.
Under THIAMINE (VITAMIN B-i) DEFICIENCY, The Merck Manual (1992), you will find that Bi deficiency is commonly caused by dextrose and alcohol as well as eating white rice. You will also find that Harvey Wiley, MD, in The History of a Crime Against the Food Law (1929), warned the food industry against using dextrose, or corn sugar, in any of its forms as a sweetener and preservative in processed food because he found that it encouraged generalized “debility” in the young men who ate canned food with dextrose used as sugar. In digestion, any refined sugar can be converted into alcohol, then acetaldehyde, by the GI tract’s intestinal flora. Although Dr. Wiley didn’t know that mechanism, the Merck Manual’s description of vitamin B1 pathology should give everyone cause to worry: “The most advanced neural changes occur in the peripheral nerves, particularly of the legs. The distal segments are characteristically affected earliest and most severely. Degeneration of the medullary sheath has been demonstrated in all tracts of the cord, especially in the anterior horn and posterior ganglion cells. Lesions of hemorrhagic polioencephalitis occur in the brain when deficiency is severe” (page 969). This is also a precise description of an acute polio virus infection, and yet it is a description of B1 vitamin deficiency caused damage. why hasn’t anyone noticed this strange coincidence? As you will read, Dr. Richard Bruno, Ph.D., found brain lesions in the hypothalamus of people who had had polio diagnosed and who died in the 1950s. He believed, and he still believes, that polio virus caused the lesions and that they were worsened by overwork and emotional stress as diagnosed polio survivors aged. However, the most obvious cause of known brain lesions is not a virus at all, but a sugar-alcohol induced B1 vitamin deficiency that used to be known as beriberi. Here the Merck Manual identifies the ominous consequence of people who may not have been damaged by a virus at all, but by an industrialized diet leading to B 1 deficiency. The normal industrialized diet of white bread, white rice, and sugar may be the major player in setting up early signs of beriberi characterized by “fatigue, irritation, poor memory, sleep disturbances,... pain, anorexia, abdominal discomfort, and constipa-tion (a Merck beriberi description)” that have been incorrectly associated with a virus infection.
All of these chronic fatigue symptoms are the usual symptoms of chronic illness: FM, PPS, MS, CFS, not to mention alcoholism, cerebral beriberi, Creutzfeldt-Jakob, Kuru, and Wernicke-Korsakoff syndrome. Full blown dry beriberi involves not only burning feet, gum problems, calf pains, leg pains, but extreme weakness of the legs that may extend to other parts of the body. In wet beriberi, heart problems arise leading to heart failure. Why hasn’t anyone noticed that heart disease and neurological problems are related? All of this is brought on by a demineralized, devitaminized diet, and may have been the basic dietary cause that led to acute paralysis and even death in many people diagnosed with acute polio infection. In fact, the virus may have been attracted by sites of pre-existing injury in the brain and spinal cord. Polio viral infection, I believe is a secondary cause of nerve damage, not a primary cause. But there are many causes. As I wrote Beating Chronic Illness, I was convinced that childhood illnesses and vaccines played another major role. Under Merck’s “Poliomyelitis,” you will not find any confirmation of that theory. But if you discovered as Tedd Koren, D.C., did, that polio infection was recorded only if you had a terribly acute case lasting more than two weeks alter 1953, and only if you had not been vaccinated, and then, it wasn’t recorded as polio at all. It was recorded as Acute viral Encephalitis or Aseptic Meningitis, from 1953 to the present. Vaccine special interests must have had very powerful effects on the popular media and the medical sources of information. Under meningitis headings, I found unexpected support for my theories of causes: On page 1473, Merck Manual, under the heading Infectious causes, I read: Viral causes: Mumps, echovirus, poliovirus, coxsackievirus, herpes simplex (cold sores), other herpes viruses, horse viruses, hepatitis, mono nucleosis [Epstein-Barr virus, also known as a herpes virus], and, [believe it or not,] HIV. Did you know that the aIDS virus is a causative agent in polio? Did you know that common childhood diseases are common causes of what used to be called polio?
Postinfectious causes: Measles [known to cause paralysis in sheep as well as other damage], rubella, varicella [a fancy word for chickenpox], smallpox, and cowpox. It is not unrealistic to think there is a direct connection between childhood illnesses and vaccination and what I call chronic illness. We might wonder why Multiple Sclerosis is listed as a non-infectious cause of meningitis formerly known as polio. They are both demyelinating conditions involving loss of the myelin sheath. They are both linked with vitamin and mineral deficiencies as you will see. Also listed under non-infectious causes are iatrogenic causes: chemo-therapeutic agents, antibiotics, dyes, lead.
Under the quiet heading vaccine reactions, you can read “MANY”[my caps], including rabies, pertussis, and smallpox. This translates: vaccines are known causes of meningitis-polio. Do you suppose your family physician has noticed this? Do you suppose anyone in the research field has noticed? I wrote one dozen international names in research to find out, but not one answered my letter.
The one Merck classification that makes a lot of sense is many drugs, including all over-the-counter non-narcotic painkillers, like aspirin and ibuprofen. And all we were told in the mass media is that aspirin was good for preventing heart disease and cancer. According to the 1993 Winter FDA Consumer report, British studies were not able to confirm the aspirin claim, and in fact found that aspirin seemed to worsen our chances of both cancer and heart disease. We might wonder how much is news report and how much on national TV is advertising that looks like news. There are many unfortunate established causes of chronic illness, and it wouldn’t hurt to know enough to help remedy the situation by eating brown rice for the natural B1 “rice polish” that cures beriberi, not synthetic B1, and all the major minerals and trace minerals we may have been deprived of most of our lives that have been missing in sugar and white bread. Chronic illness is a deficiency disease, worsened by childhood illnesses, vaccination, and many other toxic agents. Web site:http://www.mall-net.com/cooter/
Chronic Fatigue—The New Face of Polio? Just as tuberculosis, once believed to have been eradicated by modern medicine, has now returned in more virulent, drug-resistant strains, so may polio be with us again in a disguised form. We may be mistakenly calling it chronic fatigue syndrome (U.S.) or myalgic encephalomyelitis (England). According to William Campbell Douglass, M.D., editor of Second Opinion, polio is more common than ever and may actually be caused by the polio vaccination. This intriguing and potentially electrifying theory is based on information Dr. Douglass gleaned from several clinical studies.
Dr. Douglass argues that the Salk and Sabin vaccines, widely administered to children in the 1950s for poliomyelitis, did not eliminate polio at all but forced it to change its form. While the vaccines suppressed the polio virus, the virus was replaced by genetically similar ones, such as Coxsackie virus which is often found in elevated levels in chronic fatigue syndrome (CFS) patients.
The Coxsackie family of viruses, first isolated in 1948, consists of 29 different strains and is linked to numerous illnesses. When physicians first began identifying these viruses in the blood of CFS patients, they failed to discern their connection with polio. The sustained use of polio vaccines for over 40 years has resulted in “at least 72 viral strains that can cause polio-like diseases,” says Dr. Douglass. Before the polio vaccines, there were only three polio viruses. He notes that he was not the first to point to evidence of “the changing of polio rather than the elimination of it.” As early as 1934, cases of “atypical” polio were reported in Los Angeles; “abortive poliomyelitis” was reported in Switzerland in 1939.
Dr. Douglass suggests that the trend towards the emergence of a new polio—its predominant symptom changing from infantile paralysis to adult muscle weakness—has rapidly increased since the polio vaccines were introduced. “We now know that chronic fatigue syndrome is not a new disease, but simply an ‘aborted form’ of the more serious paralytic polio,” he states. If Dr. Douglass’ speculations prove correct, the credibility of conventional medicine’s mass vaccination program will be seriously undermined. It is hardly a public health benefit if a vaccine simply modifies an existing disease, forcing it to take another form in the next generation of patients. The indiscriminate use of vaccines may prove to be as counterproductive as has the overprescribing of antibiotics.
SOURCE—William Campbell Douglass, M.D., “Chronic Fatigue Syndrome: The Hidden Polio Epidemic,” Second Opinion 6:8 (August 1996), 1-6.
Regarding tonsillectomy:
“In poliomyelitis following tonsillectomy, the intervals between the operation and onset of symptoms have ranged from three to thirty days.” “By contrast the degree of paralysis in each limb of a control group of all children under three (48 in all) notified during the same period as the above 17 cases who had not received any inoculation within 35 days of the onset; and for whom exact information as to the site and severity of paralysis was ascertained, is also shown in table vi. It is clear that there is a considerable increase in the severity of the paralysis in the last-inoculated limbs of those children under three who received an injection of pertussis vaccine within thirty-five days of the onset of poliomyelitis.”
And then, the article continued onto the inevitable coverup - the inability to allow the public to know anything which would be detrimental to continued vaccination: “A report was submitted to the Chief Health Officer (Dr. G.E. Cold) on July 20, 1949, and it was decided to defer action until further evidene had been collected. When this had been done, the Chief Health Officer, in September, 1949, invited Prof. F.M. Burnet, FRS, Dr. E.V. Keogh, of the Commonwealth Serum Laboratories, and Dr. H. McLorinan, superintendent of the Infectious Diseases Hospital, Fairfield, to confer with him and officers of his department. It was agreed that there was certainly evidence of some association between prophylactic injections and development of poliomyelitis in the epidemic. This raised questions of great importance from the viewpoint of public-health administration. It was feared that immunisation, particularly against diphtheria, might be prejudiced if the public were informed. The Chief Health Officer, therefore, laid the facts and the opinions of this expert committee before the Consultative Council on Poliomyelitis of the State of Victoria for an opinion whether or not the medical profession and the public should be informed. The council recommended that doctors be advised to discontinue use of the pertussis vaccine during the currency of the epidemi, just as tonsillectomy had been postponed since it appeared to determine an attack of poliomyelitis in rare instances.” So you see, there is a causal link between both tonsillectomy and vaccination and the development of severe paralysis ( more severe than that in an unvaccinated individual. This paralysis has NOTHING to do with the polio vaccine, just with vaccination in general. As a matter of fact, it isn’t just the vaccines that can provoke polio, it is any injection.
eMail: cureimer@mb.sympatico.ca
Dr. Neustaedter, I have a couple of questions. The first is regarding the renaming of non-paralytic polio to meningitis in the 1950’s. I heard about that a few months ago. If it’s true of course the stats on polio in this country would not be accurate after that point. Do you know about this? Where could I find more about this? I’m also wondering if you can tell me when they stopped growing the polio virus on rhesus monkey kidneys. Do you believe there might be a link between AIDS and the rhesus monkey as some authors have suggested?
eMail: randalln@cris.com A full discussion of both these issues can be found in the Polio section of my book The Vaccine Guide. The criteria for diagnosing polio did change when the vaccine was introduced in the 1950s and this change in diagnosis apparently reduced the statistic for the number of polio cases dramatically. Monkey kidney cells are used to produce the oral vaccine and controversy rages about the contamination of OPV with monkey viruses. For a complete review of these issues, see the book. Here is one quote from the book. Dr. Bernard Greenberg, a biostatistics expert, was chairman of the Committee on Evaluation and Standards of the American Public Health Association during the 1950s. He testified at a panel discussion that was used as evidence for the congressional hearings on polio vaccine in 1962. During these hearings he elaborated on the problems associated with polio statistics and disputed claims for the vaccine’s effectiveness. He attributed the dramatic decline in polio cases to a change in reporting practices by physicians. Less cases were identified as polio after the vaccination for very specific reasons. “Prior to 1954 any physician who reported paralytic poliomyelitis was doing his patient a service by way of subsidizing the cost of hospitalization and was being community-minded in reporting a communicable disease. The criterion of diagnosis at that time in most health departments followed the World Health Organization definition: “Spinal paralytic poliomyelitis: signs and symptoms of nonparalytic poliomyelitis with the addition of partial or complete paralysis of one or more muscle groups, detected on two examinations at least 24 hours apart.” Note that “two examinations at least 24 hours apart” was all that was required. Laboratory confirmation and presence of residual paralysis was not required. In 1955 the criteria were changed to conform more closely to the definition used in the 1954 field trials: residual paralysis was determined 10 to 20 days after onset of illness and again 50 to 70 days after onset.... This change in definition meant that in 1955 we started reporting a new disease, namely, paralytic poliomyelitis with a longer-lasting paralysis. Furthermore, diagnostic procedures have continued to be refined. Coxsackie virus infections and aseptic meningitis have been distinguished from paralytic poliomyelitis. Prior to 1954 large numbers of these cases undoubtedly were mislabeled as paralytic poliomyelitis. Thus, simply by changes in diagnostic criteria, the number of paralytic cases was predetermined to decrease in 1955-1957, whether or not any vaccine was used.
